- Reaction score
- 18
0.5mg per day
Our persian brother got banned again, miss u bro!
#freedhtcel
Look, im not feeding anything. The version he told me was different, but im not interest in this, because we are in a hair loss fórum, soo the only thing which matters for me here is informations about the disease we share, i dont give a f*** about likes and dislikes and you guys should do the same. DHTcel had a lot of information, thats why his presence is valueable for the discussionWe live in a society and if you break the rules you're out, he's out. Some people learn from their mistakes and deserves a second chance but he's doing the same sh*t over and over and over. Am I the only one here that thinks it's weird that he engages in conversation with multiple accounts mass liking and disliking post? Instead of petting his back why don't you question him, why do you feed his behavior?
@MostoleTinker and @ikarus2 is DHTcel and he is mass disliking my posts because I dragged him on discord
You can't put an old head on young shoulders, unfortunatelyWe live in a society and if you break the rules you're out, he's out. Some people learn from their mistakes and deserves a second chance but he's doing the same sh*t over and over and over. Am I the only one here that thinks it's weird that he engages in conversation with multiple accounts mass liking and disliking post? Instead of petting his back why don't you question him, why do you feed his behavior?
This is tricky as there's evidence for both high and low estrogen linked to loss or growth.It still remains unproven whether estrogen gives results by itself or because it blocks androgens more effectively. If we believe that androgens are a common cause in both men and women, then we can say estrogen doesn't cause regrowth because of increase in its levels because women have a lot of it) but because it reduces androgens. However, bridge and other folks here believe that estrogen directly promotes hair growth. We would never know unless there are studies on it.
Another point to note is that some people experience regrowth on finasteride so we can say it's blockage of androgens, but then finasteride also increases E.
Bottom line - you can't block/decrease you androgens without increasing E (possible w cypro though) and therefore we might never know for sure.
For instance, there are studies with eunuchs showing that while their hair loss stopped upon castration, they didn't get growth without supplementing estrogen.
Umm.. maybe. When then some people like Anxiousandy, obsessive etc didn't respond to it?"ERβ knockout mice display accelerated catagen development, along with an increase in the number of apoptotic hair follicle keratinocytes (28). This suggests that, contrary to previous working hypotheses (274), ERβ does indeed play a significant role in murine hair growth control: whereas the catagen-promoting properties of E2 are mediated via ERα, ERβ may mainly function as a silencer of ERα action in hair biology (28). Nevertheless, ERα is thought to serve as the predominant ER in the hair follicle of animals (31, 233, 254)."
"Estrogens have been used for topical treatment of hair diseases for more than half a century (284) and constitute a firm staple of management strategies for female pattern androgenetic alopecia in central Europe (88). Orentreich observed in 1969 a decrease in daily effluvium during therapy with systemic estrogens (285), which were reported to increase the proliferation rate, slow down differentiation, and, thus, postpone telogen effluvium (286)."
"progesterone receptor, prolactin, and lactoferrin are examples of relevant target genes in the pilosebaceous unit with consensus EREs (263–265)."
"The number of genes that carry an ERE, whose transcription is known to be altered by E2 stimulation, is very large (294) (Table 3). Together with the fact that E2 modifies androgen metabolism and vice versa (250, 251) and the intriguing cross talk examples discussed above, this already renders the number of potential signaling cross talks and cross-modulation events that may impact how E2 can alter the growth of a given hair follicle in a defined gender and location daunting. This gets even more complicated if one enters additional recent findings into the equation. Suffice it here to list just two selected examples of this added level of complexity: possible cross talks between E2/ER and intrafollicularly generated hormones, such as melatonin (53) and prolactin (8, 9)."
Idk, some people don't respond to anything or hardly anything, even proven treatmentsUmm.. maybe. When then some people like Anxiousandy, obsessive etc didn't respond to it?
are they talking about mice?"In vitro, testosterone and estrogen inhibit hair growth, suggesting that scalp hair growth may be controlled by these sex hormones."
Haha.. I dunno what to say now.