Recent content by xRedStaRx

It's not crap, it's very legitimate. In fact, it's the best theory we have today.

I believe the way they work, and why they give permanent damage has to do with the HPA and progestrone.

Why would you want to inhibit 5α-r1 though?

Dutasteride is more potent at inhibiting type II (and type I obviously) than finasteride. Not only that, but dutasteride has a tendency to accumulate in the blood, leading to much stronger effective inhibition at tissue level, where DHT plasma levels are insignificantly putative for true...

Can we stop talking DHT now? It's missing the forest for the trees.

I assure you, he is not.

It's more about glutamate.

English must not be your first language, this is where the misunderstanding stems. I'll try to be more clear. Testosterone drops with age in statistical clinical data, but it does not 'naturally' drop with age. Hope this makes sense.

I don't know where to begin. Just accept that you are wrong. Testosterone does not naturally decline with age. It's a statistic.

No they don't. What does higher DHT levels even mean? Wrong.

The difference is that DHT is a catalyst for the process, not the problem itself. Claiming that finasteride is the best treatment we have so far does not do the pathogenesis justice. Or else we would all be NW0 by only taking finasteride, according to you. Clearly, DHT is but a small part of...

That's not true. A point that is often overlooked That's actually a good thing.

This is a great example of faulty logic and baseless conjecture.

He is right though. Why does DHT promote hair growth everywhere except some scalp hair follicles? Inhibiting DHT stops Androgenetic Alopecia, but it's not the root cause. It's primarily inflammation, autoimmunity, and disrupted calcium channeling. www.perfecthairhealth.com is an excellent source on this issue.

Finasteride will give you side effects, there is no way around it.