I'm no expert, but there is this thing called "Apoptosis", where cells basically die due to biochemical events and shrinkage is a symptom of this and it can ultimately lead to atrophy: a wasting away of part of the body.
Post-menopausal women experience a thinning of the vaginal walls. The exact mechanism of why this happens is unclear, but it probably occurs due to the body's expired need of the reproductive system. Are you then going to say that menopause is not the cause of vaginal atrophy just because the mechanisms OF the atrophy are not understood? Would you suggest that we should, instead of preventing menopause altogether, that we should focus our efforts on solving the process of atrophy, even though the only reason this atrophy is taking place at all, is directly because of the menopause?
Because that is what everyone in your camp is ultimately saying: Stop the method of the atrophy and not the thing causing it in the first place by treating the mechanism as the cause rather than the trigger for the mechanism.
The same kind of thing is obviously happening in hairloss. DHT is the biochemical event that triggers some sort of apotosis in the cells of the hair with the genetic predisposition to it.
It may very well be physically impossible to stop this process without removing the biochemical event dramatically (if not entirely) or as RepliCel's research seems to show: migrating cells immune to it, and as far as I'm aware, existing research demonstrates this to be the case.