- Reaction score
- 582
T can covert to DHT or E.
Inhibiting 5ar means more T converts to E.
High doses of E is the only thing that regrows hair. Inhibiting DHT only stops hair loss. Regrowth stories on finasteride usually involve estrogenic side effects (my own story, too), so I'd argue regrowth on 5ar-inhibitors (which is quite rare) come down to more estrogen as well.
We have the most hair when we are high T and high E, as teenagers
As our DHT (and Estrogen) declines with age, our hair declines.
Are we missing something, instead of having too much of something?
Is inhibiting 5-ar systemically merely the best way of adressing intrafollicular estrogen, by wiping out estrogens main competitor, DHT?
Inhibiting 5ar means more T converts to E.
High doses of E is the only thing that regrows hair. Inhibiting DHT only stops hair loss. Regrowth stories on finasteride usually involve estrogenic side effects (my own story, too), so I'd argue regrowth on 5ar-inhibitors (which is quite rare) come down to more estrogen as well.
We have the most hair when we are high T and high E, as teenagers
As our DHT (and Estrogen) declines with age, our hair declines.
Are we missing something, instead of having too much of something?
Is inhibiting 5-ar systemically merely the best way of adressing intrafollicular estrogen, by wiping out estrogens main competitor, DHT?
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