Based on their unimpressive results there are Wnt agonists already on the market that will meet or exceed SM's potential. Instead of waiting for SM just use VPA, it's probably more potent.
Topical valproic acid increases the hair count in male patients with androgenetic alopecia: a randomized, comparative, clinical feasibility study using phototrichogram analysis - PubMed
Valproic acid (VPA), a widely used anticonvulsant, inhibits glycogen synthase kinase 3β and activates the Wnt/β-catenin pathway, which is associated with hair growth cycle and anagen induction. To assess the efficacy of topical VPA for treating androgenetic alopecia (Androgenetic Alopecia), we performed a...pubmed.ncbi.nlm.nih.gov
If you aren't a responder to minoxidil you can take oral minoxidil, which has a 100% response rate, or get minoxidil sulfate powder and mix it yourself, also 100% response rate. Microneedling and tretinoin also turn minoxidil non-responders into responders. Or you can go the PGF2a route, latanoprost and bimatoprost are already on the market and equivalent to minoxidil. SM will not perform as well as minoxidil so it's pretty much useless. If you get side effects from minoxidil or pgf2a analogues then just use VPA instead of waiting for SM. There's no drug more advanced than minoxidil coming in the next 5 years, but you can get KY19382 on the grey market which might exceed minoxidil.
Oh wow, I had no idea Valproic acid worked as well as it does for a readily available wnt agonist. Although I don't know anyone who actively uses it? Is there any reason to why it's not more widely used?