TransradarSuspect...
Check out itsnoah story if you haven't already. This is the only example I knowIt's up to you the reason why do it. People just point out this cuz it might help you come to terms with feeling comfortable in your own skin and do what you do.
I myself believe won't bring up this topic anymore, I don't want to be annoying and you've already given your answer.
I still don't have the answers as to what would happen if I do cycles of estrogen and then test. I need to research more but I need money first so it's pointless.
This is possible with HRT. Dutasteride does not reduce DHT in the scalp sufficiently. Bicalutamide, in theory, should prevent DHT from binding to receptors at an adequate dosage, but it did not help meHas anyone lowered their scalp DHT above 90% is that possible?
I think those that are unresponsive to extensive androgenic alopecia treatments likely have a different mechanism of hairloss than androgens.Error - Cookies Turned Off
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Now I'll ruin your mood
Bicalutamide is effective in only 53% of cases of female pattern baldness. Unsurprisingly, at least half of those who tried it didn't help.
HRT may not help either, because even on HRT there will be low levels of T and DHT, and if you did not respond to Bicalutamide, you will simply continue to go bald even at the female hormonal level. Nothing can be done about it. If I have not answered Bicalutamide and am balding on the back of my head, it is quite possible that HRT will not give me anything We cannot completely get rid of androgens, and blocking the receptors does not always work. Thus, we do not have a medicine that is guaranteed to help
Spironolactone and CPA are even less effective than Bicalutamide, so the percentages are even lower in their case. Add to this the fact that Androgenetic Alopecia has more than just androgenic mechanisms. In general, we will never defeat this disease
Androgenetic Alopecia has different mechanisms, but it would be better if they were only androgens: the rest of the mechanisms are unknown to us. Apparently if the genetics are too bad and you are programmed to go bald no matter what, this is a sentenceI think those that are unresponsive to extensive androgenic alopecia treatments likely have a different mechanism of hairloss than androgens.
Estrogen does not have a concentrated topical effect. You can just as well use HRT and Tamoxifen, which will not relieve you of feminization, but can harm your hair.@Almas what would happen if somebody injects strogen in their scalp and then takes aromatasa inhibitor. Like what if the majority of strogen your body is getting is through those scalp injections? Is it possible to avoid unwanted side effects this way?
I need to research and make a thread to ask these questions if there's anybody who bothers to say anything it's gonna be appreciated.
The reason why this doesn't work is because you have to have the substrate (aromatizable androgens) and the machinery (aromatase) to produce the estrogens right where they're needed. This is the only way to get concentrations that are high enough without having to inject a pretty serious amount of estrogens. The ability to produce locally on demand means that you can have high concentrations of steroids without affecting the serum concentrations of those steroids.
I've personally found that the healthier you are, the higher both 5AR expression and aromatase are.In your opinion, how can you upregulate this process locally? No matter how crazy or dangerous it could be.
I'm going to be trying it soon. I'm working on getting WAY-200070 sourced. WAY is relatively new. I'm not sure how long 8b-vinylestradiol has been around. Neither of these are necessary easy to get ahold of, though. They're not prescription drugs... They're compounds under research and development. So, you have to find a lab to synthesize them.Is there a reason why nobody has tried this?
Thanks for your time and answers you're one of the users that I always follow.
I remember on a thread a while back we discussed this somewhere. I forget the name, but there is an experimental synthetic estrogen modified to have a way higher affinity for ER-b, and someone managed to find a source, but it was like ten thousand dollars for a gram or something lol.Is there a reason why nobody has tried this?
Thanks for your time and answers you're one of the users that I always follow.
How the results have changed since you added Bicalutamide (100mg per day, as I understand it). What do you think plays a decisive role in your regime?Months since I’ve been around.
5mg oral minoxidil (15 months 5mg-40mg)
50mg bic (3/4 months)
0.5 dust (3 yrs)
estro gel eod 1.5mg
50mg per day as I said above. Mate what plays a diverse role is not complaining about it and f*****g around with dosages all the time. Go outside, enjoy the sun, socialise, laugh, not constantly putting yourself down over hair, life’s too short.How the results have changed since you added Bicalutamide (100mg per day, as I understand it). What do you think plays a decisive role in your regime?
I am skeptical about estrogel at such low dosages. Are you applying it to your head or trying to lower your T level? I thought a lot about microdosing estradiol and came to the conclusion that it is useless, because even HRT gives poor results.