Again, it is shown that androgens trigger NO synthesis. This leads me to believe that people with Androgenetic Alopecia/male pattern baldness, if DHT is the culprit (which is supported by the fact that finasteride and dutasteride do work), should actually have high NO levels.
On the other hand, applying minoxidil also raises NO levels but is beneficial in treating male pattern baldness... this is just so confusing.
We would really, really need a biotech or chem student or grad. He could analyse scalp dermis and blood of male pattern baldness sufferers for NO levels from a) bald areas, b) balding areas, and c) healthy areas, compare them, and also compare the NO levels to blood of a person without Androgenetic Alopecia/male pattern baldness.
The role of NO is so confusing; we really really need some blood values to draw any conclusions.
The fact that locally applied NO works "antiandrogen" is not very surprising, given that it works as a vasodilator and thus flushes any accumulated DHT away.
On the other hand, applying minoxidil also raises NO levels but is beneficial in treating male pattern baldness... this is just so confusing.
We would really, really need a biotech or chem student or grad. He could analyse scalp dermis and blood of male pattern baldness sufferers for NO levels from a) bald areas, b) balding areas, and c) healthy areas, compare them, and also compare the NO levels to blood of a person without Androgenetic Alopecia/male pattern baldness.
The role of NO is so confusing; we really really need some blood values to draw any conclusions.
The fact that locally applied NO works "antiandrogen" is not very surprising, given that it works as a vasodilator and thus flushes any accumulated DHT away.