Here I will publish my results on 25 mg spironolactone.

[Yar]

According to my observations, caffeine stimulates testosterone production, while alcohol reduces it.

 

[Yar]

My sperm has become watery, I still have erections in the morning, the musky smell of sweat is gone, and my feet don't smell anymore either.

 

[Yar]

We continue treatment for androgenic alopecia with spironolactone 25 mg. Photo 1 shows a very good response to the drug, hair lengthening, hairline straightening. Photo 2 shows a lesser response to the drug, but also shows results.

 

[Yar]

I also excluded coffee from my diet because it increases androgenic activity, the skin becomes more oily in the forehead area and the hair becomes oily.

 

[bsmith]

We continue treatment for androgenic alopecia with spironolactone 25 mg. Photo 1 shows a very good response to the drug, hair lengthening, hairline straightening. Photo 2 shows a lesser response to the drug, but also shows results.

Your hair density looks good in that photo.

 

[Yar]

Your hair density looks good in that photo.

Thank you

 

[Armando Jose]

testosterone causes baldness

Scalp hairs, years before puberty, make testosterone

 

[Yar]

Scalp hairs, years before puberty, make testosterone

As soon as puberty begins, high testosterone levels begin

 

[bsmith]

I just use Finasteride and Minoxidil. That does the job for me.

That should be enough for androgen alopecia, that's really all you need.

If you're Caucasion, you have a higher follicle density, and you will go balder sooner than a non Caucasion person.

(Why is spell check marking Caucasion? does anybody know, I think that's how it's spelled).

 

[Yar]

I just use Finasteride and Minoxidil. That does the job for me.

That should be enough for androgen alopecia, that's really all you need.

If you're Caucasion, you have a higher follicle density, and you will go balder sooner than a non Caucasion person.

(Why is spell check marking Caucasion? does anybody know, I think that's how it's spelled).

Testosterone receptors need to be blocked. A single cell can have many such receptors, but only one estrogen receptor. Many transgender men and women have achieved very good results by taking spironolactone + E2. E2 will be present in the blood and create estrogen receptors in the cell. Meanwhile, spironolactone will block testosterone receptors.

 

[Yar]

formation of a new hairline

 

[exwhyyou]

I just use Finasteride and Minoxidil. That does the job for me.

That should be enough for androgen alopecia, that's really all you need.

If you're Caucasion, you have a higher follicle density, and you will go balder sooner than a non Caucasion person.

(Why is spell check marking Caucasion? does anybody know, I think that's how it's spelled).

just coz finasteride and min are enough for your standards doesnt mean thats how we all feel

 

[strictly4myhair]

Scalp hairs, years before puberty, make testosterone

As soon as puberty begins, high testosterone levels begin

So I first want to say sorry to @Yar for interrupting his thread but I'll try to make it short and will be making my own thread soon.

So heres my theory regarding Androgenetic Alopecia. Androgenetic Alopecia dosent start with Androgens(Dht/Testosterone) but it instead starts with this phenotype gene The FokI-ff. This genotype refers to a specific genetic variation in the Vitamin D Receptor (VDR) gene. In this variation, the protein is slightly longer than the standard "FF" version. People with the 'ff' genotype may have an altered response to vitamin D, which can influence bone health and immune regulation. So easily put because this gene is causes issues with how how our body responds to active vitamin d3 (calcitrol) which means immune issues and bone issues such as (Rickets, arthritis, osteoporosis etc)

Associations with this gene can cause issues such as Metabolic Syndrome, pcos, insulin resistance/diabetes, craniofacial / skeletal issues such as scoliosis, spine and jaw development isuse that @Mitko1 talks about a lot (i know a lot of people call his theory b.s. but I wouldnt say it is, hes trying to figure out hairloss and regrow his hair just like us and remember issues with skeletal health isnt external it can be internal too). Not only that this gene is associated to foxa2 and pax1 (which are skeletal issues) and credits to this user on bald truth for making this association back in 2015. https://www.baldtruthtaik.com/forum...enetic-reasons-why-we-balding-men-are-balding

So this gene (FokI - ff) is basically causing not only immune issues but it's the reason of Low Mineral Bone density too meaning risk for bone fractures and skeletal issues is high. This gene was common back then and is becoming dangerously more common now.

"Why is it common for us men to bald" well here it is, the cascade starts when puberty happens due to more androgens being introduced. So because we of Foki - ff being a phenotype defect within the vitamin d gene androgens senses we are vulnerable and basically increases bone mineral density way beyond skeletal threshold but that's to protect us from Low Bone Mineral Density complications meaning fractures, rickets, rheumatoid arthritis, osteoporosis etc. Androgens can absolutely increase Bone Mineral Density and that's a fact. So us being androgen dominant (having way more test and dht compared to women) and this alarmingly becoming - common gene. This is why men are prone to balding.

Look at Eunuchs and Castratis for example
Or even trans individuals, but specifically Castratis and Eunuchs never bald at all due and if they are balding And got castrated it stopped (Listen I'm not saying snip the nuts or chemically castrate hell nah I'm only saying this to make it click together). Because they did castrate it was well known that they had skeletal issues and it's for 2 reasons.

1. The most obvious one they had no primary hormone. "dosent this defeat the idea of your theory?" Yes you could say that but look at it like this yes absolutely for skeletal health Estrogen can help maintain bones but that's it it dosent increase density not as much as androgens at least. Estrogen does it in a delicate sense! Androgens and if you have the FokI-ff phenotype androgens will increase Bone Mineral Density AGGRESSIVELY as a way to prevent skeletal issues.

2. The FokI-ff gene itself: so while yes Castratis and Eunuchs will maintain hair and will halt balding if it started via puberty. But obviously (at least for us cis males) castration is not the way to go. YES you will grow hair and won't have to worry about balding no more - you will open yourself to a skeletal issue 100%.

So to not make this thread longer it goes like this

The FokI (ff) genotype (Vitamin d3/calcium absorption and activation issues regardless of where levels are) which can lead to other skeletal gene and health issues. > Puberty (androgens) senses the lack there of vitamin d absorption/activation. > Balding starts (for some it's aggressive for some it's slow but one things for certain balding didn't start when you turned 35 it started when puberty started)

@Armando Jose @Yar you guys inspired me to finally put what I believe to be the true cause of androgenetic alopecia out there!

 

[Yar]

So I first want to say sorry to @Yar for interrupting his thread but I'll try to make it short and will be making my own thread soon.

So heres my theory regarding Androgenetic Alopecia. Androgenetic Alopecia dosent start with Androgens(Dht/Testosterone) but it instead starts with this phenotype gene The FokI-ff. This genotype refers to a specific genetic variation in the Vitamin D Receptor (VDR) gene. In this variation, the protein is slightly longer than the standard "FF" version. People with the 'ff' genotype may have an altered response to vitamin D, which can influence bone health and immune regulation. So easily put because this gene is causes issues with how how our body responds to active vitamin d3 (calcitrol) which means immune issues and bone issues such as (Rickets, arthritis, osteoporosis etc)

Associations with this gene can cause issues such as Metabolic Syndrome, pcos, insulin resistance/diabetes, craniofacial / skeletal issues such as scoliosis, spine and jaw development isuse that @Mitko1 talks about a lot (i know a lot of people call his theory b.s. but I wouldnt say it is, hes trying to figure out hairloss and regrow his hair just like us and remember issues with skeletal health isnt external it can be internal too). Not only that this gene is associated to foxa2 and pax1 (which are skeletal issues) and credits to this user on bald truth for making this association back in 2015. https://www.baldtruthtaik.com/forum...enetic-reasons-why-we-balding-men-are-balding

So this gene (FokI - ff) is basically causing not only immune issues but it's the reason of Low Mineral Bone density too meaning risk for bone fractures and skeletal issues is high. This gene was common back then and is becoming dangerously more common now.

"Why is it common for us men to bald" well here it is, the cascade starts when puberty happens due to more androgens being introduced. So because we of Foki - ff being a phenotype defect within the vitamin d gene androgens senses we are vulnerable and basically increases bone mineral density way beyond skeletal threshold but that's to protect us from Low Bone Mineral Density complications meaning fractures, rickets, rheumatoid arthritis, osteoporosis etc. Androgens can absolutely increase Bone Mineral Density and that's a fact. So us being androgen dominant (having way more test and dht compared to women) and this alarmingly becoming - common gene. This is why men are prone to balding.

Look at Eunuchs and Castratis for example
Or even trans individuals, but specifically Castratis and Eunuchs never bald at all due and if they are balding And got castrated it stopped (Listen I'm not saying snip the nuts or chemically castrate hell nah I'm only saying this to make it click together). Because they did castrate it was well known that they had skeletal issues and it's for 2 reasons.

1. The most obvious one they had no primary hormone. "dosent this defeat the idea of your theory?" Yes you could say that but look at it like this yes absolutely for skeletal health Estrogen can help maintain bones but that's it it dosent increase density not as much as androgens at least. Estrogen does it in a delicate sense! Androgens and if you have the FokI-ff phenotype androgens will increase Bone Mineral Density AGGRESSIVELY as a way to prevent skeletal issues.

2. The FokI-ff gene itself: so while yes Castratis and Eunuchs will maintain hair and will halt balding if it started via puberty. But obviously (at least for us cis males) castration is not the way to go. YES you will grow hair and won't have to worry about balding no more - you will open yourself to a skeletal issue 100%.

So to not make this thread longer it goes like this

The FokI (ff) genotype (Vitamin d3/calcium absorption and activation issues regardless of where levels are) which can lead to other skeletal gene and health issues. > Puberty (androgens) senses the lack there of vitamin d absorption/activation. > Balding starts (for some it's aggressive for some it's slow but one things for certain balding didn't start when you turned 35 it started when puberty started)

@Armando Jose @Yar you guys inspired me to finally put what I believe to be the true cause of androgenetic alopecia out there!

Good afternoon! For example, when I'm taking finasteride, I'm starting to get shooting pains in my lower back, problems with my lower back joints, and terrible pain. I've restarted the medication twice, and both times it's been confirmed.

 

[strictly4myhair]

Good afternoon! For example, when I'm taking finasteride, I'm starting to get shooting pains in my lower back, problems with my lower back joints, and terrible pain. I've restarted the medication twice, and both times it's been confirmed.

Damn! So another thing do want to tell everybody who's on either 5ar blockers or Testosterone blockers, DEFINITELY have your Bone Mineral Density tested via deca scans and what not!

 

[bsmith]

just coz finasteride and min are enough for your standards doesnt mean thats how we all feel

I just think Spirolactane is dangerous. I understand that he is taking it topically, and not that much will enter his bloodstream.

I'm not judging anybody. I even tried using topical Estroidal (Estrogen) on my scalp, but I quit after a month because I was warned against it.

I also know transgender people who were already bald before from androgen alopecia, and their hair never grew back.