I assert it because most miniaturisation in the recipient can be attributed to concurrent miniaturisation in the donor and without more information to go off we can only assume that his case is no different.
Until there is substantive evidence that proves calcification, fibrosis, and poor blood flow have any impact on the mechanisms that cause miniaturisation during catagen and next anagen it should be disregarded as a concern. We know donor hair follicle cells have substantially less 5ar activity when compared to Norwood hairs and this is most likely why they are less susceptible to miniaturisation.