Recent content by Ein

3 or 3.5

I'm not sure about my crown. I don't think I've regained all of my density there, but my top and hairline looks fine. I've been on Bicalutamide for 21 months.

I started seeing regrowth around the eighth month mark. I have mild, yet visible crown thinning (which appears as a dreaded back part in my long hair). The density on the top was also pretty less, but I'm not sure if it was because my genes or hair loss. The parting on the top and around the...

Yes, and surprisingly, 75% of the hair that I shed are from my nape and donor area, 25% from the top and crown and 0% from the hairline and temples.

It needs to be delivered specifically to the dermal papillae and nowhere else to make sure it's safe. Now, how do you ensure that your tiny tiny amount is just delivered to the hair follicles and nowhere else? Also, I don't think that the complex is available anywhere because it's potentially...

Lol, I meant it could completely disfigured your skin, cause skin cancer or make you die even on a single exposure.

But you said that it goes away and then comes back, right? (Could be some unexpected form of Telogen Effluvium) Also, there are tons of ways of having hairloss on different parts of body. An expressed gene shows effects under normal circumstances. You can't expect to have the same facial hair on...

Yes, it is possible, but I'm not sure, because it might even differ with morphology of cells. Because the levels of androgens are in traces, there is a possibility that it binds somewhere else. Estrogen can also affect SHBG, so the traces of androgens may not just affect females. By making a...

It is useful. It works by an entirely different mechanism and reducing DHT separately is useful because it's binding capacity is much more than testosterone.

It varies depending upon what area you're talking about. 50mg/day can block almost all of it (>93%) in the skin and hair. But the same blocks around 0% receptors on the gonads.

Yes, about those three ways: The blockade of receptors is in no way comparable to that of bicalutamide. It is a steroidal selective androgen receptor antagonist. It works only by reducing the concentration of testosterone so that kinetics don't mess up with its binding. That's why it works only...

Not for long.

Masculine traits are a result of two types of receptor binding to androgen molecules. In the first type, the receptors don't have any effect on the gene expression via any method and has more of a temporary androgen dependent effect, like Muscle Development, because it's easier to store...

Lol. Dude, I'm a computer science engineering student in an IIT. I have nothing to do with science of genetics and endocrinology. But after studying about all of this, I've realized that this is not a science which is very difficult to comprehend and understand tu its fullest. These are just...

Fat distribution is not a permanent effect of androgens. Permanent effects include bone development including facial bones like brow ridge, facial and body hair distribution and growth, face shape and features like nose and eyebrows, elongation and development of phallus, pubic hair, VOICE, etc...