Men: stop eating aromatase!

[OverMachoGrande]

Body fat does serve a purpose, it keeps us warm in the winter months, it stores vitamins like vitamin-E, D, and A, it also supplies our body with a concentrated sources of calories in times of famine. But the down side is that body fat contains an enzyme called aromatase.

Aromatase has the ability to transform testosterone into estrogen. Estrogen has a nasty effects on men. First of all, estrogen has feminizing effects on men; it makes them less confident, less aggressive, less dominant, less muscular, estrogen accelerates the fusing of bone-growth-plates; making men's bone thin and short.

Researchers use to think that prostate enlargement was caused by androgens, but this did not make sense because young men have very high levels of androgens and no prostate problems (or baldness), so researchers shifted the blame from androgens to estrogens as being the real culprit behind prostate enlargement.

So, since we know that body fat contains very high levels of aromatase, why on earth would we eat animal fat. Wouldn't animal fat contain the same large amounts of aromatase?

Hero Ori Hoffmekler, author of the book: The Anti-Estrogenic Diet, claims that xenoestrogens are behind the feminization of men...but I don't think he realizes that animal fat is loaded with aromatase enzymes.

So since we know that animal fat is loaded with aromatase enzymes, what do you think would happen if you ate animal fat? You would increase estrogen levels*.

When estrogen levels rise, your body stops production of testosterone due to the feedback loop... you see, since estrogen is made from testosterone, the body senses the high estrogen and says to itself "I have too much estrogen...so I'd better stop production of testosterone, that way it cannot be converted into more estrogen".

So men...If you don't want to have clogged arteries, if you don't want to be feminized, If you don't want to have an enlarged prostate and a non-existent sex life, you will completely cut animal fats out of your diet. The largest source of animal fat is dairy products, the second source is obviously meats; cows, chickens, fish, turkey, etc.

Studies have also shown that vegan men have 13% higher testosterone levels compared to lacto-ovo-vegetarians (Allen NE. Hormones and diet: low insulin-like growth factor-I but normal bioavailable androgens in vegan men. Br J Cancer. 2000 Jul;83(1):95-7.)


*1. Rose DP, Boyar AP, Cohen C, Strong LE. Effect of a low-fat diet on hormone levels in women with cystic breast disease. 1. Serum steroids and gonadotropins. J Natl Cancer Inst 1987;78(4):623-6.

2. Ingram DM, Bennett FC, Willcox D, de Klerk N. Effect of low-fat diet on female sex hormone levels. J Natl Cancer Inst 1987;79:1225-9.

3. Goldin BR, Gorbach SL. Effect of diet on the plasma levels, metabolism and excretion of estrogens. Am J Clin Nutr 1988;48:787-90.

 

[OverMachoGrande]

misterE,

Great post, I want to add a couple of important things as well;

Not only is fat a precursor to higher estrogen levels, but also the precursor to the "big three" correlated diseases. The fat can be very harmful as;

a) Fat builds up in the arteries and veins restricting blood flow, causing high LDL cholesterol, promoting arterial plaque, driving the individual to increased chances of developing heart disease.

b) The excess estrogen being converted from fat pollutes the liver and 'water' system, in turn, effecting the health of the prostate, causing it to enlarge, and further, effecting the healthy function of the prostate, resulting in the changing of androgen/enzyme/estrogen production. Also, through toxifying the water system (mainly kidneys), the kidneys fail to do their job in cleansing the blood of these same substances.

c) Fat diets, especially high fat diets we have here in the states, assist in causing inflammation to the epithelial cells, thus, fats helping bring the onset of diabetes through inflammation.

I also want to note that not only do we not need nearly as many fats in todays day and age, but the fats we do consume, are cooked at very high tempetures, so even great, "heart healthy" fats like olive oil, become quite deadly as all the micro-nutrients are converted into highly toxic substances, which further increases inflammation.

Did I mention many middle aged men, and those suffering from male pattern baldness have higher estrogen and lower testosterone than women. :shock:

So, basically it would a great idea to cut most if not all fats out of the diet (especially since we've accumulated quite a lot over the years guys), and if you want to keep any fats in your diet, get them from an avacado occasionally, or a few seeds, maybe a couple of nuts .

misterE, I am surprised this info has never been mainstreamed, you would think this would be vital in consideration to male pattern baldness, keep it up man!

 

[OverMachoGrande]

Not only is fat a precursor to higher estrogen levels, but also the precursor to the "big three" correlated diseases.

That's right.
Dietary fat clogs the arteries.
Dietary fat causes insulin resistance.
Dietary fat increases estrogen levels which then stimulates the prostate.

I also want to note that not only do we not need nearly as many fats in todays day and age, but the fats we do consume, are cooked at very high tempetures, so even great, "heart healthy" fats like olive oil, become quite deadly as all the micro-nutrients are converted into highly toxic substances, which further increases inflammation.

Bingo!

So, basically it would a great idea to cut most if not all fats out of the diet (especially since we've accumulated quite a lot over the years guys), and if you want to keep any fats in your diet, get them from an avacado occasionally, or a few seeds, maybe a couple of nuts .

Yes, I believe that the lower the fat, the better. Hero John McDougall always says: "The type of fat you eat is the type of fat you wear".

Nuts, seeds, and avocado should be considered a delicacy; eaten on rare occasions. Eating raw vegetables will provide you with all the essential-fatty-acids you would ever need and with the right percentage. Fat should be kept below 10 or 15% of your total calorie intake.

misterE, I am surprised this info has never been mainstreamed,

Unfortunately I'm not.

 

[Bryan]

When estrogen levels rise, your body stops production of testosterone due to the feedback loop... you see, since estrogen is made from testosterone, the body senses the high estrogen and says to itself "I have too much estrogen...so I'd better stop production of testosterone, that way it cannot be converted into more estrogen".

Well, so you're finally beginning to learn a little about the HPT axis! Remember just a year or so ago when you were constantly making the claim that the body responds to estrogen by making more DHT? Remember how I had to tell you CONSTANTLY (maybe a dozen times) that estrogen reduces the synthesis of androgens, but you simply wouldn't hear of it? Congratulations on finally getting a little bit smarter!

 

[baller234]

[quote="misterE] [/i]estrogen accelerates the fusing of bone-growth-plates making men's bone thin and short.
[/quote]

You're right, estrogen does accelerate the EGP fusion. In fact it's really the only reason we stop growing. Men with aromatase deficiency have been reported to continue to grow taller into their late 20s. However, estradiol is also THE MOST potent promoter of bone growth. Think of it as a double edged sword, it speeds up the rate of growth drastically (estrogen is the reason we have "growth spurts" during puberty) but it also causes the EGPs to "burn out" faster. Also, it is well known that estrogen is very benefical for bone density. I'm not sure if you were stating otherwise when you said it makes them thin.

 

[shootingguard]

I gotta say man, I don't really understand :dunno:

 

[OverMachoGrande]

When humans eat the body-fat of animals they are eating aromatase enzymes (because body-fat contains high levels of aromatase). As a result their levels of estrogens rise in their body.

 

[dougfunny]

Even worse I bet is all of the synthetic hormones and crap that they fed the cows to make them grow that now go into you!

 

[Bryan]

When humans eat the body-fat of animals they are eating aromatase enzymes (because body-fat contains high levels of aromatase). As a result their levels of estrogens rise in their body.

Oh, so you think that aromatase is active orally? What a wonderully innovative and creative hypothesis! uke:

 

[OverMachoGrande]

Even worse I bet is all of the synthetic hormones and crap that they fed the cows to make them grow that now go into you!

:agree:

No kidding, have you seen Food Inc., or read The Food Revolution?

And to think, You are what you eat.

 

[raj47]

i am having high triglycerites level in blood and vldl and ldl also in high level
past from one month i am taking oats as my main meal ..

 

[dougfunny]

i am having high triglycerites level in blood and vldl and ldl also in high level
past from one month i am taking oats as my main meal ..

fyi taking 3-4 grams of fish oil daily lowers triglycerides around 40%. It's one of the very few supplements that actually has sound scientific data backing it up.

 

[JLL]

Dietary fat clogs the arteries.

No.

Am J Clin Nutr. 2004 Nov;80(5):1102-3.
...In multivariate analyses, a higher saturated fat intake was associated with a smaller decline in mean minimal coronary diameter (P = 0.001) and less progression of coronary stenosis (P = 0.002) during follow-up. Compared with a 0.22-mm decline in the lowest quartile of intake, there was a 0.10-mm decline in the second quartile (P = 0.002), a 0.07-mm decline in the third quartile (P = 0.002), and no decline in the fourth quartile (P < 0.001); P for trend = 0.001. .... Monounsaturated and total fat intakes were not associated with progression. CONCLUSIONS: In postmenopausal women with relatively low total fat intake, a greater saturated fat intake is associated with less progression of coronary atherosclerosis, whereas carbohydrate intake is associated with a greater progression.

PMID: 15531663 [PubMed - indexed for MEDLINE]

 

[47thin]

Jim Fixx, may he rest in peace, died at 49, eating a high carb, low fat diet.

The head of the raw food movement, whose name escapes me, lived to the ripe old age of 73!! Wow, 73, 7 years less than my still alive, total junk food, tv dinner eating mom, who is going to be 81! Is she healthy!? Hell no! But she's not dead, either! And at least she enjoys what she eats!

 

[OverMachoGrande]

If you eat animal fat, you are eating aromatase enzymes. As a consequence, your estrogen levels increase, lowering your testosterone production...leaving DHT to become the dominate androgen (due to it's high affinity to the androgen receptor/SHBG).

 

[Bryan]

If you eat animal fat, you are eating aromatase enzymes. As a consequence, your estrogen levels increase...

Here's your next homework assignment: find some medical or scientific evidence that aromatase is active when consumed orally. There: THAT oughtta keep you busy for awhile!

 

[OverMachoGrande]

It makes sense, If you eat aromatase your estrogen levels increase. Body fat is aromatase, rather it be in a human or animal.

 

[moxsom]

Researchers use to think that prostate enlargement was caused by androgens, but this did not make sense because young men have very high levels of androgens and no prostate problems (or baldness), so researchers shifted the blame from androgens to estrogens as being the real culprit behind prostate enlargement.

Your thinking here is flawed. Try again....

 

[OverMachoGrande]

Researchers use to think that prostate enlargement was caused by androgens, but this did not make sense because young men have very high levels of androgens and no prostate problems (or baldness), so researchers shifted the blame from androgens to estrogens as being the real culprit behind prostate enlargement.

Your thinking here is flawed. Try again....

Oh really, what do you make of this?


Estradiol Activates the Prostate Androgen Receptor and Prostate-specific Antigen Secretion through the Intermediacy of Sex Hormone-binding Globulin


Atif M. Nakhla, Nicholas A. Romas and William Rosner.

Abstract

These experiments were designed to examine the relationship between the effects of steroid hormones mediated by classic intracellular steroid hormone receptors and those mediated by a signaling system subserved at the plasma membrane by a receptor for sex hormone-binding globulin. It is known that unliganded sex hormone-binding globulin (SHBG) binds to a receptor (RSHBG) on prostate membranes. The RSHBG·SHBG complex is rapidly activated by estradiol to stimulate adenylate cyclase, with a resultant increase in intracellular cAMP. In this paper we examine the effect of this system on a prostate gene product known to be activated by androgens, prostate-specific antigen. In serum-free organ culture of human prostates, dihydrotestosterone caused an increase in prostate specific antigen secretion. This event was blocked by the anti-androgens cyproterone acetate and hydroxyflutamide. In the absence of androgens, estradiol added to prostate tissue, whose RSHBG was occupied by SHBG, reproduced the results seen with dihydrotestosterone. Neither estradiol alone nor SHBG alone duplicated these effects. The estradiol·SHBG-induced increase in prostate-specific antigen was not blocked by anti-estrogens, but was blocked both by anti-androgens and a steroid (2-methoxyestradiol) that prevents the binding of estradiol to SHBG. Furthermore, an inhibitor of protein kinase A prevented the estradiol·SHBG-induced increase in prostate-specific antigen but not that which followed dihydrotestosterone. These data indicate that there is a signaling system that amalgamates steroid-initiated intracellular events with steroid-dependent occurrences generated at the cell membrane and that the latter signaling system proceeds by a pathway that involves protein kinase A.

http://www.jbc.org/content/272/11/6838.abstract

 

[moxsom]

umm that paper says that Androgen's do play a role in prostate enlargement.