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'Abstract
In the dental industry, it seems no one has entertained the thought that if a dislocated mandible, in a Class II Skeletal relation could occlude or block the blood flow in in the STA [Superior Temporal Artery]. Therefore stopping or impeding blood flow to the vertex of the head and therefor causing balding. Taking into consideration that in a healthy Class I Skeletal relation, the clearance between the base of the skull and the eminence of the condyle is only 3.5 mm. Orthodontic experience tells us that mandibles 3.5 mm retro gnathic and more are very common. The intent of this paper is to expose the facts and evidence that show the Class II Skeletal mandible is the cause of balding by blocking blood flow through the S.T.A. balding of vertex can be avoided. A large array of research has been, and continues to be conducted to determine the causative agent for hair loss. Traditionally such investigation has focused on a number of varying topics, including but not limited to chromosome composition, genotype, and subsequent phenotype expression. However, little or no investigation has been conducted to deduce what effect the skeletal relationship has upon the initial development of hair loss. Through a series of observational case studies, it is evident that there is a relationship between malocclusion and hair loss. Specifically, through analysis of dental records, cephalometric radiographs, and visual observation of patients, there appears to be a correlation between Class II Skeletal malocclusion and subsequent hair loss. Further investigation yields that vascular anatomical differences between different skeletal schemes is associated with the development of hair loss.
It is clear that the Superior Temporal Artery (STA) [1-4] represents 85% of the vascular supply to the scalp, whilst the remaining 15% is provided by the Occipital Artery (OA). In healthy Class I skeletal relationship (Figure 1), there is adequate clearance (3.5 mm) between the base of the skull, and the eminence of the condyle, to allow for adequate perfusion of the scalp by the STA [5]. However, in the instance of Class II Skeletal malocclusion, there is the possibility of reduced perfusion or occluded blood flow in the STA, due to the retrognathic architecture of the skull. Evaluation of the population reveals that hair loss is seen overwhelmingly among individuals with Class II skeletal profile, supporting the hypothesis that malocclusion leading to reduced vascular perfusion of the scalp in turn results in hair loss (Figure 2).'
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In the dental industry, it seems no one has entertained the thought that if a dislocated mandible, in a Class II Skeletal relation could occlude or block the blood flow in in the STA [Superior Temporal Artery]. Therefore stopping or impeding blood flow to the vertex of the head and therefor causing balding. Taking into consideration that in a healthy Class I Skeletal relation, the clearance between the base of the skull and the eminence of the condyle is only 3.5 mm. Orthodontic experience tells us that mandibles 3.5 mm retro gnathic and more are very common. The intent of this paper is to expose the facts and evidence that show the Class II Skeletal mandible is the cause of balding by blocking blood flow through the S.T.A. balding of vertex can be avoided. A large array of research has been, and continues to be conducted to determine the causative agent for hair loss. Traditionally such investigation has focused on a number of varying topics, including but not limited to chromosome composition, genotype, and subsequent phenotype expression. However, little or no investigation has been conducted to deduce what effect the skeletal relationship has upon the initial development of hair loss. Through a series of observational case studies, it is evident that there is a relationship between malocclusion and hair loss. Specifically, through analysis of dental records, cephalometric radiographs, and visual observation of patients, there appears to be a correlation between Class II Skeletal malocclusion and subsequent hair loss. Further investigation yields that vascular anatomical differences between different skeletal schemes is associated with the development of hair loss.
Introduction
Despite overwhelming evidence highlighting the importance of vascular perfusion to the scalp in order to circumvent hair loss, little research has been conducted regarding the varying anatomical vascular landscape of the scalp. Namely, there are differences in the vascular anatomical landscape of the scalp, given varying occlusal schemes.It is clear that the Superior Temporal Artery (STA) [1-4] represents 85% of the vascular supply to the scalp, whilst the remaining 15% is provided by the Occipital Artery (OA). In healthy Class I skeletal relationship (Figure 1), there is adequate clearance (3.5 mm) between the base of the skull, and the eminence of the condyle, to allow for adequate perfusion of the scalp by the STA [5]. However, in the instance of Class II Skeletal malocclusion, there is the possibility of reduced perfusion or occluded blood flow in the STA, due to the retrognathic architecture of the skull. Evaluation of the population reveals that hair loss is seen overwhelmingly among individuals with Class II skeletal profile, supporting the hypothesis that malocclusion leading to reduced vascular perfusion of the scalp in turn results in hair loss (Figure 2).'
Malocclusion and Hair Loss: An Intimate Relationship
Malocclusion and Hair Loss: An Intimate Relationship, Brian Dye*
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