I haven't.
I've seen graphs were the efficacy was shown to be marginally less for lower dosage, but not much. So you could try different dosages for yourself.
But making decisions based of one or two studies is a bit iffy.
Big bone is probably referring to this studyhttps://onlinelibrary.wiley.com/doi/full/10.2164/jandrol.108.005025
Their conclusion:
In this review, we surveyed studies on erectile dysfunction in patients treated with 5 α‐reductase inhibitors and critically examined the evidence that associates 5 α‐reductase inhibitors and erectile dysfunction. We conclude that 5 α‐reductase inhibitors do not lead to erectile dysfunction to a significant degree, and we support the position that dihydrotestosterone is less relevant than testosterone in erectile function.
^That study was a sort of meta analysis. So to understand it fully you need to go through each cited study and look at the mean age population. You'll see that there are statistical problems with their inclusion criteria.
Again everyone can quote studies back and forth until we're all sick. But just look at the biochemistry, anatomy and pathological process of DHT and the prostate gland. DHT functions as a secondary sex hormone, allowing for full development of male genetalia at puberty. and the prostate gland functions primarely to produce seminal fluid. DHT also has systemic effects and prostate gland is also responsible for
A collection of nerves that is located in the fascia covering the prostate is named the prostatic plexus. They arise from the lower (inferior) portion of the pelvic plexus and are distributed not only to the prostate but also to the corpora cavernosa of the penis and urethra. Injury or damage to these nerves impacts on the mechanisms of erection and can thus cause erectile dysfunction
Further if you remove your prostate gland, you will have dramatic negative effects on penile tissue. See:
https://synapse.koreamed.org/search...534/wjmh.2017.35.1.1&code=2074WJMH&vmode=FULL
"In addition to urinary incontinence and erectile dysfunction, several other impairments of sexual function potentially occurring after radical prostatectomy (RP) have been described;"
Overall I agree that in the studies, ED did not seem to be a serious complication to people taking finasteride, it was slightly higher than a placebo effect. And I do not want to discount the studies outright, there were double blind, controlled studies with thousands of users. But I do believe the studies underreported sexual side effects because they relied on self assessment and because their mean age groups were middle age and older.
I personally put more credence in the physiologic process of the body than studies.