Does Trt Make Hair Loss Worse? Low T?
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It could because more T means more DHT. And DHT is causing baldness. I got off creatine because theres a study that says it increases DHT levels by 50%
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It seems there is a correlation between younger men losing hair and having LOW testosterone and low SHBG and LOWER serum DHT. So why then would men with low blood levels of androgens suffer from an androgen related disorder? Most likely because the body in response to low circulating androgens will increase tissue production of potent androgens to make up for the lack of blood androgens (such as DHT in the hair and prostate) causing tissue specific androgen disorders.
TrT should only be started if you have primary hypogonadism, if you have secondary you should try to find the cause of the low T. Usually there is no identifiable cause so taking something like Clomid is a start.
Healthy production of testosterone and estrogen is good for hair growth however simply injecting testosterone may accelerate hair loss if your body has already increased the rate of T to DHT and has not shut that off when more T is introduced.
This is not a good advice. Clomid is a bad method to increase testosterone because
1-it increases SHBG .. so although total testosterone increases, the free testosterone does not..
2- intratesticular aromatisation increases estrogen levels which will counter the effect of testosterone.
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You do not know what you are talking about. Taking testosterone will also increase SHBG. Clomid raises sperm count AND free testosterone.
https://www.ncbi.nlm.nih.gov/pubmed/12904801
The study was performed to measure the impact of testosterone (T) administration on circulating levels of 5alpha-dihydrotestosterone (DHT). Group 1 (32 men; mean age 61 years; mean T 6.9 +/- 1.9 nmol l(-1)) were treated for 15 months with long-acting T undecanoate. Group 2 (23 men, mean age 60 years, mean T 7.6 +/- 2.0 nmol l(-1)) were treated for 9 months with T gel. Plasma T and DHT were measured before and after 9 months T administration. In the men treated with T undecanoate plasma T and DHT were also measured after 12 and 15 months. Before T administration, plasma DHT ranged from 0.39 to 1.76 nmol l(-1) (0.30-1.90 nmol l(-1)). Mean DHT declined upon T administration from 0.95 +/- 0.50 to 0.55 +/- 0.30 nmol l(-1) (P < 0.05). With an arbitrary cut-off at 0.60 nmol l(-1), all 21 values of DHT > 0.60 nmol l(-1) had fallen from 1.29 +/- 0.50 to 0.70 +/- 0.60 nmol l(-1) (P < 0.01). Below this cut-off point 13 values rose and 21 fell upon T administration. Below this cut-off point values on average declined from 0.39 +/- 0.12 to 0.30 +/- 0.14 nmol l(-1) (P < 0.05). The study revealed that in a cohort of elderly men with subnormal plasma T levels plasma DHT levels declined upon T administration when they were in the higher range of normal (>0.6 nmol l(-1)), with a profound shift of DHT/T ratios presumed to be an indicator of a reduced 5alpha-reductase activity. Below plasma DHT levels of 0.6 nmol
Abstract
The effect of clomiphene citrate (CG) on sex hormone binding globulin (SHBG) was studied in 10 oligozoospermic patients with varicocele and 6 normospermic men. Plasma SHBG, testosterone (T), oestradiol (E2), FSH, LH, Prolactin (Prl), thyroxine (T4) and 17-OH-progesterone (17-OH-P) were determined before and during medication. SHBG concentration rose from 38.1 +/- 18.3 to 54.3 +/- 16.0 nmol/l (P less than 0.01), while T and E2 showed significant increases from 31.2 +/- 10.8 nmol/l and 24.6 +/- 5.4 pg/ml to 52.0 +/- 3.6 and 43.3 +/- 14.9, respectively in the oligozoospermic patients, with similar rises noted in the normospermic men. FSH, LH and 17-OH-P were markedly elevated while on CC, but Prl and T4 remained unchanged. The findings of this study indicated the CC causes an increase of SHBG concentration, which is probably related to the rise of E2 concentration. This SHBG change, combined with the intrinsic oestrogenic activity of CC might be one of the factors responsible, through a decrease of free T and a T to E2 imbalance, for the lack of significant effect on parameters of seminal quality in so treated patients.
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What are you talking about? You are posting random crap to try and prove that Clomid does not increase free testosterone when it clearly does.
Are you blind?