Could someone tell me the typical scalp and serum testosterone levels and DHT levels before and after various med doses? Any info would help. I'm trying to figure out how much of the testostosterone spike from Avodart is from the brain telling the testes to make more, and how much is from 5ar in the scalp not turing the testosterone into DHT.
This is important because it will tell us if we are mainly keeping testosterone from turning into DHT, or dumping 10 testosterones into the scalp for every DHT kept out.
Serum levels are helpful because they are a good measure of what the testes are cranking out. Is it possible for extra testosterone to get to the scalp without also rasing body levels as high? I read that avodart raises scalp testosterone levels 104%, and the 2.5mg/day 154%, and the proscar about 20%, though I could be wrong on the 20%. How accurate are those figures likely.
Bryan says increase teste's activity is the primary cause, but how is it possible for the scalp levels to climb so much when the serum levels do not?
Docj077 found that in some men, or some androgen receptors, testosterone may have the same affinity for the AR as DHT does, and would do the same thing DHT does. This could explain why men tend to reciend in the hair line before balding in the vertex, and why propecia tends to be more effective in back than in front. Maybe the front tends to not care as much what androgen lands on it, where as the back is only hurt by DHT. Maybe raising scalp levels are not such a good idea. If the brain telling the testes to make more testosterone is the main cause, then maybe a more moderate avodart dose that raises testosterone only 20% would be better, and a topical avodart or GTE would be good for locally preventing testosterone converstion one to one. Bryan's latest topical study showed topical finasteride regrowing hair in front. Perhaps this is because serum testosterone levels are not raised. This would also support that study I thought I saw. In a study Bryan posted recently, a chemical blocked DHT better than it did testosterone, which Bryan said was an unexpected result. I wonder if it was an experimental fluke, or a genetic difference. I'm not too worried about testosterone because I put Eucapil on my front.
I realize this is speculation, but if someone would find those androgen blood concentration levels so we can find out if there is more of a one to one exchange or a systemic rise from the testes or a sharp rise just in the scalp, that could really shed some light. I'd also like some guesses at how accurate the info is.
This is important because it will tell us if we are mainly keeping testosterone from turning into DHT, or dumping 10 testosterones into the scalp for every DHT kept out.
Serum levels are helpful because they are a good measure of what the testes are cranking out. Is it possible for extra testosterone to get to the scalp without also rasing body levels as high? I read that avodart raises scalp testosterone levels 104%, and the 2.5mg/day 154%, and the proscar about 20%, though I could be wrong on the 20%. How accurate are those figures likely.
Bryan says increase teste's activity is the primary cause, but how is it possible for the scalp levels to climb so much when the serum levels do not?
Docj077 found that in some men, or some androgen receptors, testosterone may have the same affinity for the AR as DHT does, and would do the same thing DHT does. This could explain why men tend to reciend in the hair line before balding in the vertex, and why propecia tends to be more effective in back than in front. Maybe the front tends to not care as much what androgen lands on it, where as the back is only hurt by DHT. Maybe raising scalp levels are not such a good idea. If the brain telling the testes to make more testosterone is the main cause, then maybe a more moderate avodart dose that raises testosterone only 20% would be better, and a topical avodart or GTE would be good for locally preventing testosterone converstion one to one. Bryan's latest topical study showed topical finasteride regrowing hair in front. Perhaps this is because serum testosterone levels are not raised. This would also support that study I thought I saw. In a study Bryan posted recently, a chemical blocked DHT better than it did testosterone, which Bryan said was an unexpected result. I wonder if it was an experimental fluke, or a genetic difference. I'm not too worried about testosterone because I put Eucapil on my front.
I realize this is speculation, but if someone would find those androgen blood concentration levels so we can find out if there is more of a one to one exchange or a systemic rise from the testes or a sharp rise just in the scalp, that could really shed some light. I'd also like some guesses at how accurate the info is.
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