LinuxCavalier
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I don't think there is enough acell discussion here.
people seem to think that acell may be great from hair transplant donor area perspective.. why does there seem to be little interest in a follica-type wounding + acell method?
if we assume that hair loss is caused by changes in the follicle itself (precipitated by androgen exposure) as opposed to changes in the surrounding tissues, then what is to stop new follicle formation via acell? Acell certainly seems to have created new follicles in wounded animals.
I'd also like to build on BornThisWay's post about Laminin-511 (also called Laminin-10. The 511 refers to alpha chain type 5, beta chain type 1, gamma chain type 1). Laminin is an extracellular matrix protein that apparently signals embryonic stem cells to start growing hair. ACell, an extracellular matrix product, contains large amounts of Laminin. I would love to know if it specifically if ACell contains Laminin-10, but can't find much about it.
people seem to think that acell may be great from hair transplant donor area perspective.. why does there seem to be little interest in a follica-type wounding + acell method?
if we assume that hair loss is caused by changes in the follicle itself (precipitated by androgen exposure) as opposed to changes in the surrounding tissues, then what is to stop new follicle formation via acell? Acell certainly seems to have created new follicles in wounded animals.
I'd also like to build on BornThisWay's post about Laminin-511 (also called Laminin-10. The 511 refers to alpha chain type 5, beta chain type 1, gamma chain type 1). Laminin is an extracellular matrix protein that apparently signals embryonic stem cells to start growing hair. ACell, an extracellular matrix product, contains large amounts of Laminin. I would love to know if it specifically if ACell contains Laminin-10, but can't find much about it.
