I HAVE A CONCERN ABOUT BLOCKING ESTROGEN.

[Guest]

I just got a prescription for Arimidex.

I plan on taking 0.5mg every 5 days to prevent Gyno, weight gain.

BRYAN maybe you can shed some light.

Will be result in an upregulation of AR.. so that when Im off Arimidex, I might have an influx of estrogen in my body? More than before?

 

[amsch]

It may help to open up some more topics about making fun of the Dutasteride side effects, just to be cool! :bravo:
viewtopic.php?f=46&t=49200

 

[$tackz]

I'm not understanding why people on this forum opt to take aromatase inhibitors as opposed to a selective estrogen receptor modulator that would simply bind to the receptor instead of affecting free estrogen throughout the body. Seems like a safer choice as far as your endocrine system goes in the long run.

 

[Timi]

what is with Progesteroncreame?

is that better by Östrogendominance?

with dutasteride i have lost my Facial features
my Bodyskin like pudding

take today finasteride only all 2Days i hope my Body Restore

Timi

 

[Bryan]

I just got a prescription for Arimidex.
I plan on taking 0.5mg every 5 days to prevent Gyno, weight gain.

Will be result in an upregulation of AR.. so that when Im off Arimidex, I might have an influx of estrogen in my body? More than before?

By "AR", are you referring to androgen receptors? I haven't heard of Arimidex having an effect on androgen receptors, but if anybody else knows better, please post it here. What Arimidex certainly WILL do, of course, is upregulate the production of testosterone, although that won't be too significant at the low dose you'll be using.

When you stop using Arimidex you'll certainly have a mild "influx of estrogen", but just as a result of no longer using an aromatase inhibitor. As far as I know there's no rebound effect, if that's what you're hinting at. Again, if anybody knows differently, please post it here.

 

[Bryan]

I'm not understanding why people on this forum opt to take aromatase inhibitors as opposed to a selective estrogen receptor modulator that would simply bind to the receptor instead of affecting free estrogen throughout the body. Seems like a safer choice as far as your endocrine system goes in the long run.

Why does that sound like a safer choice to you? Assuming that estrogen does exert its effects on body tissues through the estrogen receptor, I can't think of any a priori reason why the receptor blocker would be any "safer" than the enzyme inhibitor (or vice versa). Lacking any direct scientific evidence to the contrary, it seems to me to be six of one, half a dozen of the other.