I want to know if its topical 5ar2 inhibiting ability is compared to internal finasteride.
I know from the study that it is not an androgen receptor blocker, and that it does inhibit 5ar1, but I don't know if it inhibits 5ar2.
The study said it is stronger than the other fatty acids, but did not say how strong it is, at least not in the abstract. I realize it would be very difficult to measure 5ar1 and DHT levels. But I was wondering if Bryan read the full study, and knows how much GLA was used and how much is shrank the sebum glands compared to other stuff and egcg studies.
I know from the study that it is not an androgen receptor blocker, and that it does inhibit 5ar1, but I don't know if it inhibits 5ar2.
The study said it is stronger than the other fatty acids, but did not say how strong it is, at least not in the abstract. I realize it would be very difficult to measure 5ar1 and DHT levels. But I was wondering if Bryan read the full study, and knows how much GLA was used and how much is shrank the sebum glands compared to other stuff and egcg studies.