Recent content by yanez

I would like to start a topical cyproterone acetate treatment can anyone suggest me the concentration usually used?

I would like to start a topical dutasteride treatment can anyone suggest me the concentration usually used?

I have taken it it's the third times I apply it I have noticed these sides effects a slight brain fog ..like if I had drunk a glass of whisky and sometimes a slight sense of nausea but rarely

Thanks so I belived Fluridil was a androgen receptors blocker ..then now I discover it works on degrading androgen receptors.. very interesting ..it works like ASC-J9 in theory http://www.hairsite2.com/flu.pdf http://www.ncbi.nlm.nih.gov/pubmed/12174057 http://findarticles.com/p/articles/mi_m...

thnx

so you mean you belive that the follicles reparation is faster from the relief of not being attacked by androgens (when the receptors are blocked) rather than they degenerate from exposure to androgens (when the receptors are uncovered ) " So you think could be enough more than 50% relief in...

I have wrote to Rambo Lee of Koutingchem he told me Their current price is USD 200 for 200MG Then send me some documents I link here http://rapidshare.com/files/452484295/1 ... 05_ftp.pdf http://rapidshare.com/files/452484336/2008.pdf http://rapidshare.com/files/452484375/ASC_J9.pdf...

ok I have read this viewtopic.php?f=23&t=64003&st=0&sk=t&sd=a&start=450 from haarlijn and this from baldinglikeamofo viewtopic.php?f=23&t=64003&st=0&sk=t&sd=a&start=470 so people belive even with AR blocker upregulation could be a problem ..above all when AR blockers let receptors...

Thanks all for suggestions guys :) :bravo:

have you the link of this study ? thanks in advance :)

hi have you studies that confirm these theories ? @enden ok thanks ,are your assumptions or have you studies that confirm what you say Thanks bye

ok Thanks a lot anyway I think that with normal AR blocker (spironolactone,flutamide,fluridil,ciproterone) ASC-J9 could remain a good integration now I have to inform better on Ru and CB :)

I don't understand well this I think follicle remain unprotected it's a big problem have you studies that consider these theories? I don't understand neither this..if the bind is strong and no receptor born before the last ones die ..what's the problem to having a binded AR ? sorry maybe...

I don't know well these AR blocker ( RU58841 or CB-03-01) ..but I knew that the main problem of androgen receptor blocker is that they can keep blocked the receptor only for a while ,then the receptor reply itself and it's no more keep blocked ..so using ASC-J9 if this will not degrade all...

ok yes true I thought about those "normal "androgen blocker like ciproterone or spironolactone or fluridil that don't armour the follicle from every androgens. then anyway with these that armour the follicle fina is useless too no?