Why Does Balding Wasn't A Thing 20 Years Ago?

Haironnu

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only reason people think we are balding more now than 20 years ago is because balding wasn't discussed as much as it does now.

we have the internet, social media, we have forums, we have youtubers, to put the spot light on balding, so of course it puts more light on the whole subject, and balding people are more aware of it now, back then you did not have all that.
you didn't have club meetings of baldies discussing how much balding sucks, and you didn't have instagram to scrolls through thousands of pictures of people and examine their scalps, so of course you didn't "see" many young guys balding.

until someone actually shows a study that shows we are balding earlier now more than in the past, then everything is the same as it was, we are simply more aware of it now.
 

baba_yaga

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Desperation leads people to place hope into such things. But it is not an excuse to become delusional or ignorant.
 

pegasus2

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@baba_yaga this image- https://i.imgur.com/2Go9Q5N.jpg reminded me of this thread.

both of them are basically walking skeletons due to starvation, but still have close to full head of hair, so much for "diet can cause/accelerate hair loss" lmao

If anything that picture is anecdotal support of the idea that diet has an impact on hair loss. It's not lack of nutrition that contributes to hair loss, but over-nutrition. Diabetes and hair loss are connected. Fasting induces autophagy and improves insulin sensitivity, both of which are good for hair. You "hair loss is 100% genetic" folks misunderstand us when we talk about diet. We are not saying diet is the cause of hair loss. High sugar diets are a contributing factor. Now someone is going to post a picture of a fat guy with a full head of hair as if that means anything lol

Just keep ignoring the study I already posted that states hair loss is no more than 80% genetic.

only reason people think we are balding more now than 20 years ago is because balding wasn't discussed as much as it does now.

we have the internet, social media, we have forums, we have youtubers, to put the spot light on balding, so of course it puts more light on the whole subject, and balding people are more aware of it now, back then you did not have all that.
you didn't have club meetings of baldies discussing how much balding sucks, and you didn't have instagram to scrolls through thousands of pictures of people and examine their scalps, so of course you didn't "see" many young guys balding.

until someone actually shows a study that shows we are balding earlier now more than in the past, then everything is the same as it was, we are simply more aware of it now.

I already posted one earlier in the thread. That's the problem with these threads, it doesn't matter how much evidence you post, people continue to come in and spew the same ignorant comments.

You cant be serious...

It's not his only avenue for treating hair loss. I'm pretty sure he's using minoxidil and finasteride as well. Avoiding sugar and carbs can't hurt your hair, but even if it doesn't slow down hair loss it's going to help your overall health, so why shouldn't he do it?

Here's another study that will be ignored by the closed-minded:

Androgenetic alopecia, metabolic syndrome, and insulin resistance: Is there any association? A case–control study

Our results showed that MS-positive cases tended to have an early-onset Androgenetic Alopecia, an observation previously reported.[33] This finding may support the hypothesis that early Androgenetic Alopecia could be a clinical marker of IR.[8] Whether IR induces or promotes Androgenetic Alopecia needs to be clarified by further studies. Therefore, cases with early-onset Androgenetic Alopecia should be assessed for components of MS and IR for early detection and control of cardiovascular risk factors.

In summary, MS is significantly associated with Androgenetic Alopecia particularly, early-onset alopecia. IR is mostly the underlying pathologic mechanism. More prospective studies are required in order to objectively clarify whether the increased risk of CHD in Androgenetic Alopecia can be attributed to dyslipidemia due to androgens, IR alone, or MS due to IR. Early-onset Androgenetic Alopecia patients should be closely followed-up in the long term, particularly for CHD. These data may raise awareness in susceptible individuals that lifestyle changes (weight control, exercise, and eating meals with a low glycemic index) in early life can reduce the risk of CHD.

Conclusion:
Our results support the recommendation for assessing MS and IR in all young males with stage III or higher Androgenetic Alopecia. Early intervention is critical to reduce the risk and complications of cardiovascular disease and type 2 diabetes mellitus later in life.


If you believe that diet is something that shouldn't be considered in Androgenetic Alopecia patients then you must also believe it shouldn't be considered for preventing diabetes or cardiovascular disease. They are all genetic diseases, and some people will never get them no matter what their diet looks like, but I'm sure all you guys know better than the medical specialists at the AHA and ADA.

Judicious Toggling of mTOR Activity to Combat Insulin Resistance and Cancer: Current Evidence and Perspectives

When mTORC1 is chronically activated following excessive glucose or branched chain amino acid consumption, the sustained simulation of downstream S6K1 increases IRS1 Ser307 phosphorylation. This decreases its activity and responsiveness to insulin, thereby rendering the cell insulin resistant

Stimulation of Hair Growth by Small Molecules that Activate Autophagy
mTOR and AMPK modulation by rapamycin, metformin, and α-KG induces anagen hair growth

We report that quiescent (telogen) hair follicles can be stimulated to initiate anagen and hair growth by small molecules that activate autophagy, including the metabolites α-ketoglutarate (α-KG) and α-ketobutyrate (α-KB), and the prescription drugs rapamycin and metformin, which impinge on mTOR and AMPK signaling.


You want mTOR activity for hair, but overactivation of it, such as in diabetics, is harmful.
 
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Haironnu

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@pegasus2 end of day if you have Androgenetic Alopecia eating cleaner will not stop it nor significantly slow it down, especially if it's aggressive, there is no reason to deny yourself from pleasure/comfort foods because you have Androgenetic Alopecia. granted I'm not saying your entire diet should consist of sugar and junkfood, but completely avoiding them in hopes that your hair will stop shedding is ridiculous.
 

baba_yaga

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Avoiding sugar and carbs can't hurt your hair, but even if it doesn't slow down hair loss it's going to help your overall health, so why shouldn't he do it?
Agreed, but he shouldnt say as it is slowing down his hair loss where there isnt any concrete evidence that it certainly slows it down. This is misleading to newcomers and gives them false hope.
 

pegasus2

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Agreed, but he shouldnt say as it is slowing down his hair loss where there isnt any concrete evidence that it certainly slows it down. This is misleading to newcomers and gives them false hope.

Nobody is going to stop or reverse Androgenetic Alopecia with diet and exercise alone. Slow things down though, yeah. If you're pre-diabetic and you start eating healthy and get into shape, it's probably going to help keep your hair loss from accelerating faster. It's not going to replace meds like finasteride and minoxidil. Nothing will.

Anyone care to explain why 5 out of 11 twin pairs in this study had differences in hair volume despite having the same genetics? How is this possible when epigenetics doesn't contribute to hair loss? If it was 1 out of 11 then maybe you could argue that it's a slight possibility that the minute number of genetic mutations just happened to fall on genes that are responsible for Androgenetic Alopecia. With 5 out of 11 pairs being different even the most deluded person can't make that argument with a straight face.
https://www.researchgate.net/public...igenetic_differences_in_androgenetic_alopecia

In one pair Androgenetic Alopecia onset happened six years earlier in one twin. In another pair, one twin was a NW3 while the other a NW5. This despite the fact that identical twins often live very similar lifestyles. Even small changes in environment can seemingly trigger significantly different outcomes.

This is not to say that hair loss is caused by anything talked about in this thread, but that it can be triggered early or accelerated.

Identical twins before and after treatement. I guess the twin on top got better genes.
1.PNG
 
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baba_yaga

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Nobody is going to stop or reverse Androgenetic Alopecia with diet and exercise alone. Slow things down though, yeah. If you're pre-diabetic and you start eating healthy and get into shape, it's probably going to help keep your hair loss from accelerating faster. It's not going to replace meds like finasteride and minoxidil. Nothing will.

Anyone care to explain why 5 out of 11 twin pairs in this study had differences in hair volume despite having the same genetics? How is this possible when epigenetics doesn't contribute to hair loss? If it was 1 out of 11 then maybe you could argue that it's a slight possibility that the minute number of genetic mutations just happened to fall on genes that are responsible for Androgenetic Alopecia. With 5 out of 11 pairs being different even the most deluded person can't make that argument with a straight face.
https://www.researchgate.net/public...igenetic_differences_in_androgenetic_alopecia

In one pair Androgenetic Alopecia onset happened six years earlier in one twin. In another pair, one twin was a NW3 while the other a NW5. This despite the fact that identical twins often live very similar lifestyles. Even small changes in environment can seemingly trigger significantly different outcomes.

This is not to say that hair loss is caused by anything talked about in this thread, but that it can be triggered early or accelerated.

Identical twins before and after treatement. I guess the twin on top got better genes.
View attachment 142234
This is very interesting. Something is at play (in addition to genes) for sure.
 

Niki99

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Nobody is going to stop or reverse Androgenetic Alopecia with diet and exercise alone. Slow things down though, yeah. If you're pre-diabetic and you start eating healthy and get into shape, it's probably going to help keep your hair loss from accelerating faster. It's not going to replace meds like finasteride and minoxidil. Nothing will.

Anyone care to explain why 5 out of 11 twin pairs in this study had differences in hair volume despite having the same genetics? How is this possible when epigenetics doesn't contribute to hair loss? If it was 1 out of 11 then maybe you could argue that it's a slight possibility that the minute number of genetic mutations just happened to fall on genes that are responsible for Androgenetic Alopecia. With 5 out of 11 pairs being different even the most deluded person can't make that argument with a straight face.
https://www.researchgate.net/public...igenetic_differences_in_androgenetic_alopecia

In one pair Androgenetic Alopecia onset happened six years earlier in one twin. In another pair, one twin was a NW3 while the other a NW5. This despite the fact that identical twins often live very similar lifestyles. Even small changes in environment can seemingly trigger significantly different outcomes.

This is not to say that hair loss is caused by anything talked about in this thread, but that it can be triggered early or accelerated.

Identical twins before and after treatement. I guess the twin on top got better genes.
View attachment 142234
With that I agree. I don't think genetics is 100% the reason but It sounded like you think genetics isn't even the biggest factor at first tbh.
 

WaccWaccWacc

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Nobody is going to stop or reverse Androgenetic Alopecia with diet and exercise alone. Slow things down though, yeah. If you're pre-diabetic and you start eating healthy and get into shape, it's probably going to help keep your hair loss from accelerating faster. It's not going to replace meds like finasteride and minoxidil. Nothing will.

Anyone care to explain why 5 out of 11 twin pairs in this study had differences in hair volume despite having the same genetics? How is this possible when epigenetics doesn't contribute to hair loss? If it was 1 out of 11 then maybe you could argue that it's a slight possibility that the minute number of genetic mutations just happened to fall on genes that are responsible for Androgenetic Alopecia. With 5 out of 11 pairs being different even the most deluded person can't make that argument with a straight face.
https://www.researchgate.net/public...igenetic_differences_in_androgenetic_alopecia

In one pair Androgenetic Alopecia onset happened six years earlier in one twin. In another pair, one twin was a NW3 while the other a NW5. This despite the fact that identical twins often live very similar lifestyles. Even small changes in environment can seemingly trigger significantly different outcomes.

This is not to say that hair loss is caused by anything talked about in this thread, but that it can be triggered early or accelerated.

Identical twins before and after treatement. I guess the twin on top got better genes.
View attachment 142234

I didn’t read the whole study, but as a Statistics graduate a sample size of 11 is very unsuitable for any scientific claim. You can argue this all you want and bash on me, it’ll just show your statistical understanding.

nevertheless, could you explain the study? Why do your photos show an increase of hair at the 12 month mark? Is this a finasteride study?

I don’t have time to write walls on reasons this study shows little importance and why 5/11 could indeed be insignificant a lot of times - I have finals I am studying for but give me till the weekend to actually read the study. What I did find though in the chart I have attached is that your extreme example displays very agressive-prone hairloss as case 11 both progressed from NW1 to a NW5&NW3v in 4 years, also in this case both members held the same alcohol and smoking habits.

like I said, im quite busy but Ill get back to this on the weekend.
 

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WaccWaccWacc

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How obtuse can you possibly be? This isn't a poll, and it's not some predictive correlational study. It's just to show if twins can have different onset of hair loss despite having the same genes. For that purpose a sample size of 11 is as good as 11,000 when 45% have different rates of hair loss. It's not enough of a sample to predict the rate at which hair loss in twins will differ or the environmental causes, but it's enough to confirm that identical twins can have different rates of hair loss, which is all I need to prove my point. I guess you'd need to be shot 11,000 times to know if being shot makes you bleed. You're really desperate to deny reality and maintain your delusions. Come back this weekend and post all you want, I'm done with this thread. I don't have time for this.

“For that purpose a sample size of 11 is as good as 11,000 when 45% have different rates of hair loss.“

You post evidence but have no analytical skills. I’m guessing you’re an english major. Just read that quote, go to anyone with a bachelors degree in statistics and tell them that. They’ll laugh at you. 11 is a handful of people, say 4/11 showed “significant differences” you 45% nearly half is now reduced to 36 nearly a third.

Stop making claims with these weak *** studies, it’s honestly bad.

look how passionate you feel about your “findings”, you just stated 11 is as good as 11,000... you cannot be serious. Blind activism. You’d post anything that’s relevant to your side, even some article written on a blogpage by a high schooler.

edit: nobody is saying its 100% genetics. You just go around these forums posting wack *** polls/studies and dislike people’s posts that counter you with legitimate facts. You hold no credibility besides a mouth and a peanut brain. Explains why one would try to go from a NW2 to a NW1 at the risk of cancer. Lmao, waste of space.
 
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TheZyzz

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What people don't understand also is that we reached the peak of humanity, we are 8 f*****g billion, i mean isn't that making us see more balding and bald people? Take a look at how much Earth's population grew by years.

The only thing that keeps drawing my attention is the fact that i think and notice more and more younger dudes with advanced balding and from what i saw in the past, at least from my own experiences and what my eyes saw, real life, pictures bla bla, there were more rare or maybe i haven't payed so much attention to it, maybe social media and the internet is bringing it more to the light who knows, without some proper studies done on it i doubt we will ever know..


dude
look at concerts from the 90's and 2000's ...
first of all look at the band members

they are all in their 20's and have no signs of hairloss
second,
look at the crowd.
Bunch of men in their 20's and 30's.. their hair looks flawless, you can't even find one men with visible temples.

So yeah, I think I can say for sure that people nowdays are balding much more than 20 years ago for some reason.
 

TomRiddle

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dude
look at concerts from the 90's and 2000's ...
first of all look at the band members

they are all in their 20's and have no signs of hairloss
second,
look at the crowd.
Bunch of men in their 20's and 30's.. their hair looks flawless, you can't even find one men with visible temples.

So yeah, I think I can say for sure that people nowdays are balding much more than 20 years ago for some reason.

Yeah i don't know what to say man, it's a controversial subject and we don't actually have some valuable data about it. I feel the same from what i see daily on the internet and in real life also but it's not like i can make my own study to actually validate these things so whatever i would do i can't actually say that i know for sure...
 

Incinerate

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@pegasus2 You think the DHT surge which come from Masturbation is enough to cause hairloss. lol. You think every increase on DHT has an impact on your hair ?
Sorry, that's terribly stupid. Your study about masturbation increasing DHT is irrelevant to conclude about any links between masturbation/p**rn and hairloss.
 

TomRiddle

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@pegasus2 You think the DHT surge which come from Masturbation is enough to cause hairloss. lol. You think every increase on DHT has an impact on your hair ?
Sorry, that's terribly stupid. Your study about masturbation increasing DHT is irrelevant to conclude about any links between masturbation/p**rn and hairloss.

He is retarded la half of this forum is also... They read a bunch of articles and studies online and they believe things based of an analysis made by their mentally ill brains, with things taken out of context that suit their mentally ill mindset and ideologies... It's useless and just a waste of time to even give them the slightest attention, it's what they want and crave for the most, you are doing them a favor and giving them the wanted satisfaction. I have done it already to much, don't be me and learn from my mistakes...
 

TheZyzz

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another example:
look at the band members (they are 25+ years old here)
and most of all
look at the crowd
look at the guys who jump in from the crowd to the stage.. they have like a full head of hair, thick and long..

they are at their 20's..
these days even people who got hair at their 20's don't have this thickness.

It's amazing and I wish I knew what are the differences between now and 30 years ago.
 

karatekid

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another example:
look at the band members (they are 25+ years old here)
and most of all
look at the crowd
look at the guys who jump in from the crowd to the stage.. they have like a full head of hair, thick and long..

they are at their 20's..
these days even people who got hair at their 20's don't have this thickness.

It's amazing and I wish I knew what are the differences between now and 30 years ago.
Dude You cant look at a video at derive conclusions about a global scale phenomena. You just cant.
 
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